{"id":7170,"date":"2025-06-10T22:56:36","date_gmt":"2025-06-10T20:56:36","guid":{"rendered":"https:\/\/rvh-synergie.org\/actualites\/le-medicament-a-la-polyarthrite-rhumatoide-est-prometteur-pour-le-traitement-dune-inflammation-excessive-due-au-covid-19\/"},"modified":"2025-06-10T22:56:55","modified_gmt":"2025-06-10T20:56:55","slug":"le-medicament-a-la-polyarthrite-rhumatoide-est-prometteur-pour-le-traitement-dune-inflammation-excessive-due-au-covid-19","status":"publish","type":"post","link":"https:\/\/rvh-synergie.org\/actualites\/le-medicament-a-la-polyarthrite-rhumatoide-est-prometteur-pour-le-traitement-dune-inflammation-excessive-due-au-covid-19\/","title":{"rendered":"Le m\u00e9dicament \u00e0 la polyarthrite rhumato\u00efde est prometteur pour le traitement d&rsquo;une inflammation excessive due au Covid-19"},"content":{"rendered":"<div>\n<div class=\"article-gallery lightGallery\">\n<div data-thumb=\"https:\/\/scx1.b-cdn.net\/csz\/news\/tmb\/2025\/a-promising-therapeuti.jpg\" data-src=\"https:\/\/scx2.b-cdn.net\/gfx\/news\/hires\/2025\/a-promising-therapeuti.jpg\" data-sub-html=\"COVID-19 causes a severe cytokine storm leading to excessive inflammation of organs. Iguratimod\u2014a safe drug already in use for treating rheumatoid arthritis\u2014suppresses excessive inflammation caused by COVID-19 in mouse models, without reducing the antiviral immune response, leading to higher survival rates and viral clearance from the lungs. Credit: Institute of Science Tokyo\">\n<figure class=\"article-img\">\n<\/figure><\/div>\n<\/div>\n<p>La pand\u00e9mie Covid-19 caus\u00e9e par l&rsquo;infection SARS-COV-2 est marqu\u00e9e par une inflammation excessive de plusieurs organes, conduisant \u00e0 une d\u00e9faillance multi-organes. La reconnaissance du virus par notre corps d\u00e9clenche de graves r\u00e9ponses immunitaires, provoquant une \u00abtemp\u00eate de cytokines\u00bb, ce qui peut entra\u00eener des l\u00e9sions pulmonaires aigu\u00ebs et un syndrome de d\u00e9tresse respiratoire aigu\u00eb.<\/p>\n<p>Conventionnellement, les glucocortico\u00efdes (GC) sont utilis\u00e9s pour r\u00e9duire les temp\u00eates de cytokines caus\u00e9es par le Covid-19. Mais les GC suppriment \u00e9galement les r\u00e9ponses immunitaires antivirales du corps, retardent la clairance virale des poumons et aggravent la pneumonie virale chez les patients. Par cons\u00e9quent, une th\u00e9rapie anti-inflammatoire qui ne supprime pas la r\u00e9ponse immunitaire antivirale est n\u00e9cessaire.<\/p>\n<p>To address this gap, a research team led by Professor Shinsuke Yasuda and Associate Professor Tadashi Hosoya, along with graduate student Seiya Oba, from the Department of Rheumatology, Graduate School of Medical and Dental Sciences at Institute of Science Tokyo (Science Tokyo), Japan, in collaboration with the National Institute of Infectious Diseases, explored the use of an anti-rheumatic drug\u2014iguratimod (IGU) &#8211; En am\u00e9liorant la survie des souris infect\u00e9es par le SARS-COV-2.<\/p>\n<p>L&rsquo;ob\u00e9sit\u00e9 est l&rsquo;un des facteurs de risque les plus forts de Covid-19 s\u00e9v\u00e8re. L&rsquo;\u00e9quipe de recherche a rapport\u00e9 que la graisse visc\u00e9rale agit comme un r\u00e9f\u00e9rentiel de cellules inflammatoires, faisant de l&rsquo;ob\u00e9sit\u00e9 un facteur de risque significatif de Covid-19 dans une \u00e9tude pr\u00e9c\u00e9dente. Les souris ob\u00e8ses infect\u00e9es par le SRAS-COV-2 pr\u00e9sentaient un risque accru de temp\u00eate de cytokines. Par cons\u00e9quent, ils ont d\u00e9cid\u00e9 d&rsquo;utiliser des souris ob\u00e8ses infect\u00e9es par SARS-COV-2 comme mod\u00e8le de temp\u00eate de cytokines dans leur \u00e9tude actuelle. Les r\u00e9sultats ont \u00e9t\u00e9 publi\u00e9s dans le <i>Journal europ\u00e9en de pharmacologie<\/i> le 5 juin 2025.<\/p>\n<p>Des souris m\u00e2les de huit semaines B6 ont \u00e9t\u00e9 nourries d&rsquo;un rythme gras (HFD) \u00e9lev\u00e9 pendant dix semaines, apr\u00e8s quoi ils ont \u00e9t\u00e9 inject\u00e9s avec une dose sous-l\u00e9tale de SARS-CoV-2. Des traitements m\u00e9dicamenteux ont \u00e9t\u00e9 administr\u00e9s aux souris 1 heure avant l&rsquo;inoculation du virus jusqu&rsquo;\u00e0 4 jours apr\u00e8s l&rsquo;infection. Trois jours apr\u00e8s l&rsquo;infection, les poumons ont \u00e9t\u00e9 analys\u00e9s ou les souris ont \u00e9t\u00e9 surveill\u00e9es jusqu&rsquo;\u00e0 10 jours pour enregistrer les taux de survie.<\/p>\n<p>\u00ab\u00a0Nous avons \u00e9valu\u00e9 l&rsquo;efficacit\u00e9 de la dexam\u00e9thasone (DEX) &#8211; un m\u00e9dicament anti-rhumatisme utilis\u00e9 cliniquement, IGU, un inhibiteur de la r\u00e9ponse pro-inflammatoire, H-151, et un contr\u00f4le, respectivement, apr\u00e8s les avoir administr\u00e9s \u00e0 des souris HFD-B6\u00a0\u00bb, explique Yasuda les groupes de traitement exp\u00e9rimentaux de leur \u00e9tude.<\/p>\n<p>Les souris trait\u00e9es avec des IGU ont montr\u00e9 des taux de survie plus \u00e9lev\u00e9s que les autres groupes de traitement et ont d\u00e9montr\u00e9 une inflammation relativement l\u00e9g\u00e8re et des dommages pulmonaires par rapport aux autres groupes.<\/p>\n<p>Hosoya \u00e9labore davantage sur leurs r\u00e9sultats: \u00ab\u00a0Nous avons effectu\u00e9 une coloration immunohistochimique de l&rsquo;antig\u00e8ne SARS-COV-2 N dans les poumons 3 jours apr\u00e8s l&rsquo;infection pour comprendre si les IGU pouvaient supprimer l&rsquo;expansion virale dans les poumons et ont constat\u00e9 que le rapport des cellules de l&rsquo;antig\u00e8ne SARS-COV-2 diminuait significativement dans le groupe IGU.\u00a0\u00bb \u00ab\u00a0.<\/p>\n<p>L&rsquo;\u00e9quipe de recherche a \u00e9galement montr\u00e9 que chez la souris administr\u00e9e avec des IGU, les g\u00e8nes induits par l&rsquo;infection qui soutiennent l&rsquo;immunit\u00e9 antivirale n&rsquo;\u00e9taient pas supprim\u00e9s, contrairement \u00e0 ceux trait\u00e9s par dex. Ils ont observ\u00e9 que le DEX supprimait les r\u00e9ponses inflammatoires chez les souris infect\u00e9es par SARS-COV-2 tout en affaiblissant simultan\u00e9ment les r\u00e9ponses immunitaires antivirales, entra\u00eenant une r\u00e9duction des taux de survie et un mauvais pronostic dans ce groupe.<\/p>\n<p>L&rsquo;IGU est un m\u00e9dicament s\u00fbr d\u00e9j\u00e0 approuv\u00e9 pour \u00eatre utilis\u00e9 dans le traitement de la polyarthrite rhumato\u00efde. Cette \u00e9tude pointe vers son efficacit\u00e9 en tant que m\u00e9dicament immunomodulatoire pour les patients atteints de cytokines s\u00e9v\u00e8res li\u00e9es \u00e0 la Covid-19. Avec d&rsquo;autres \u00e9tudes cliniques, l&rsquo;IGU peut \u00eatre repositionn\u00e9 comme un agent th\u00e9rapeutique prometteur pour les infections SARS-COV-2.<\/p>\n<p>                                        <!-- print only --><\/p><\/div>\n","protected":false},"excerpt":{"rendered":"<p>La pand\u00e9mie Covid-19 caus\u00e9e par l&rsquo;infection SARS-COV-2 est marqu\u00e9e par une inflammation excessive de plusieurs<\/p>\n","protected":false},"author":1,"featured_media":7171,"comment_status":"closed","ping_status":"","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[86],"tags":[561,3217,284,223,974,1708,1046,2274,8,437,2275,261],"class_list":["post-7170","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-les-maladies","tag-covid19","tag-due","tag-dune","tag-est","tag-excessive","tag-inflammation","tag-medicament","tag-polyarthrite","tag-pour","tag-prometteur","tag-rhumatoide","tag-traitement","generate-columns","tablet-grid-50","mobile-grid-100","grid-parent","grid-50","resize-featured-image"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v23.4 - 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